Alcohol cessation in Visual Snow Syndrome aims to reduce neurovascular and neuroinflammatory processes that potentially exacerbate symptomatology. By discontinuing alcohol consumption, patients may experience reduced cortical hyperexcitability and improved neural network stability, potentially mitigating visual processing abnormalities associated with the syndrome. The neurological mechanisms involve modulating GABA receptor function and reducing glutamatergic excitation, which can help stabilize sensory processing pathways.