Furosemide is a loop diuretic that acts by inhibiting the Na-K-2Cl cotransporter in the thick ascending limb of the loop of Henle, leading to reduced sodium and chloride reabsorption. In the context of Visual Snow Syndrome, it may modulate neuronal excitability and cortical hypermetabolism through its potential neuromodulatory effects, potentially reducing abnormal visual processing and cortical hyperactivity associated with the syndrome.