A low carbohydrate diet in Visual Snow Syndrome may potentially modulate neuroinflammatory processes and metabolic dysfunction by reducing glucose-induced neuronal hyperexcitability and minimizing oxidative stress. The dietary intervention appears to influence neurological symptomatology through metabolic adaptation, potentially lowering glutamatergic neurotransmission and enhancing neuronal energy metabolism via ketone body production. This approach may provide a complementary neurometabolic strategy to mitigate visual snow symptoms by altering neural network excitability and inflammatory responses.